Jim Humble:
Miracle Mineral Supplement

This Breakthrough can save your life, or the life of a loved one.

Quoted from Jim Humble’s website (www.miraclemineral.org): “The answer to Bird Flu, Swine Flu, AIDS, hepatitis A, B and C, malaria, herpes, TB, most cancers and many more of mankind’s worst diseases has been found. Many diseases are now easily controlled. More that 75,000 disease victims have been included in the field tests in Africa. Scientific clinical trials have been conducted in a prison in the country of Malawi, East Africa.

Straight Talk on MMS – Guest: Andreas Kalcker

Now, in one year since the first book was published more than 200,000 Americans are using The Miracle Mineral Supplement (MMS). Hundreds of lives have been saved. Reports of overcoming incurable diseases are happening every day! More than 10,000 bottles of MMS are being sold each month. The most reliable source we have found for MMS is our sponsor www.MiracleMineral.us

The author has treated more than 6,000 people personally. This is the greatest breakthrough for all time and is bound to change medicine for ever!

Jim Humble and the Miracle Mineral

The inventor believes that this information is too important to the world that any one person or any group should have control. The FREE e-book download on Jim Humble’s site gives complete details of this discovery. Please help make sure that it gets to the world free. There are many medical facts that have been suppressed and this invention must not be added to that list. The name of the e-book is The Miracle Mineral Supplement of the 21st Century. This book tells the story of the discovery, and how to make and use it. This book could save your life. Give it a try. It is available for purchase at this Jim Humble approved maker: Miracle Mineral

Jim Humble tells the story behind Miracle Mineral Supplement(1 hr 14m)

Download Part 1 FREE

Part I of the e-book pretty much  tells everything,.  The book is now in its fourth edition.  It  gives more details and covers the data more thoroughly.

This is an image of a model of a chlorine dioxide ion (ClO2). It is the secret to this new mineral. It is the most powerful killer of disease pathogens known to mankind, and that is a known fact. Now, listen to Jim Humble as he explains a little about his discovery and his e-book.

What is Chlorine Dioxide?

Chlorine dioxide is a chemical compound with the formula ClO2. Prominent uses include water purification, oral hygiene, and more recently, oral supplementation. According to third party sources:

• Chlorine dioxide is used in many industrial water treatment applications as a biocide including cooling towers, process water and food processing.
• Chlorine dioxide was the principal agent used in the decontamination of buildings in the United States after the 2001 anthrax attacks.
• Chlorine dioxide was also used after Hurricane Katrina (2005) to eradicate dangerous mold from houses inundated by water from massive flooding.
• Chlorine dioxide is less corrosive than chlorine and superior for the control of legionella bacteria.
• Chlorine dioxide is more effective than chlorine against viruses, bacteria and protozoa, including the cysts of Giardia and the oocysts of Cryptosporidium (parasites).
• Chlorine dioxide is the topic of author, scientist, chemist and humanitarian, Jim Humble’s book entitled, “Breakthrough, The Miracle Mineral Supplement of the 21st Century“. In Breakthrough, Humble describes how he discovered the use of chlorine dioxide as an alternative treatment for Malaria, which has since led to over 75,000 documented successful treatments of the disease in Africa. Humble’s research aims to establish MMS as a powerful alternative treatment to many pathogen-borne diseases.
• Chlorine dioxide can be used to kill disease-bearing bacteria, yeasts, molds, fungi and algae, including MRSA and other deadly pathogens.

“MMS is not chlorine dioxide; MMS is sodium chlorite (NaClO2) 22.4%. Mixing with acid briefly produces chlorous acid (HClO2), which in successive steps oxidizes ambient chlorite (ClO2-) to produce chlorine dioxide (ClO2). Chlorine dioxide is the yellow gas produced in solution and diluted before use. Chlorine dioxide is a potent broad spectrum anti-microbial agent. It is true that ascorbates and other antioxidants taken the same day of treatment and any protein in the stomach at the time of treatment will react with ClO2 and render it less or ineffective.”
Dr. Thomas Hesselink, M.D.

Chlorine Dioxide and Blood Chemistry

To understand why the Miracle Mineral Supplement works one must understand some of the chemistry of chlorine dioxide and some of the chemistry of blood. Chlorine dioxide is a gas that is dissolved in water when in the body. Chlorine and chlorine dioxide have been used as disinfectants for more than a hundred years and there is little doubt that they simply destroy pathogens of all kinds. Both have been used in water purification systems for more than 50 years. In recent years, water purification systems using chlorine has been used less and instead chlorine dioxide is used a great deal more as it has many benefits over chlorine. Chlorine dioxide is used extensively in water purification systems throughout Europe. Although chlorine dioxide is somewhat more expensive than chlorine, its many benefits over chlorine has resulted in it being used more extensively in water purification systems than chlorine.

In 1998 The American Chemical Society, Analytical Chemistry Division said chlorine dioxide is the most powerful antimicrobial agent known to man.

Stabilized Oxygen, a diluted solution of sodium chlorite, diluted further with water very slowly gives off chlorine dioxide. The MMS is just a stronger solution to which a food grade acid has been added. The acid such as lemon juice or citric acid often used in soft drinks reduces the solution to an acid condition but still within a food range which releases up to about 1-ppm chlorine dioxide, a level of concentration that is sometimes found in processed food but is 100′s of times that which is produced in Stabilized Oxygen.

How does MMS (chlorine dioxide) work in the body?
Once it is introduced into the bloodstream, chlorine dioxide performs a highly energetic acceptance of four electrons when it comes across any cell that is below a pH value of 7. This means that diseased cells are essentially vaporized, or more technically, ‘oxidized’ while healthy cells are unaffected.

What is a pathogen?
A pathogen is described as “any biological agent that causes disease or illness to its host”. Types of pathogens include bacteria, viruses, protozoa, fungi, parasites and proteins. It is commonly known that pathogens cannot survive in an oxygen rich environment or a pH balanced internal environment.

Jim Humble: Plain Talk On His Book

MMS: Lyme Disease

Clinical Evaluations: Chlorine Dioxide

Article
J R Lubbers, S. Chauan, and J.R. Bianchine
Controlled clinical evaluations of chlorine dioxide, chlorite and chlorate in man.
To assess the relative safety of chronically administered chlorine water disinfectants in man, a controlled study was undertaken. The clinical evaluation was conducted in the three phases common to investigational drug studies. Phase I, a rising dose tolerance investigation, examined the acute effects of progressively increasing single doses of chlorine disinfectants to normal healthy adult male volunteers. Phase II considered the impact on normal subjects of daily ingestion of the disinfectants at a concentration of 5 mg/l. for twelve consecutive weeks.

Persons with a low level of glucose-6-phosphate dehydrogenase may be expected to be especially susceptible to oxidative stress; therefore, in Phase III, chlorite at a concentration of 5 mg/l. was administered daily for twelve consecutive weeks to a small group of potentially at-risk glucose-6-phosphate dehydrogenase-deficient subjects. Physiological impact was assessed by evaluation of a battery of qualitative and quantitative tests.

The three phases of this controlled double-blind clinical evaluation of chlorine dioxide and its potential metabolites in human male volunteer subjects were completed uneventfully. There were no obvious undesirable clinical sequellae noted by any of the participating subjects or by the observing medical team. In several cases, statistically significant trends in certain biochemical or physiological parameters were associated with treatment; however, none of these trends was judged to have physiological consequence. One cannot rule out the possibility that, over a longer treatment period, these trends might indeed achieve proportions of clinical importance. However, by the absence of detrimental physiological responses within the limits of the study, the relative safety of oral ingestion of chlorine dioxide and its metabolites, chlorite and chlorate, was demonstrated.

Abdel-Rahman MS, Couri D, Bull RJ.

Article

Kinetics of Cl02 and effects of Cl02, Cl02-, and Cl03- in drinking water on blood glutathione and hemolysis in rat and chicken.
Since chlorination of drinking water produces organochlorinated substances (some possibly carcinogenic), the use of chlorine dioxide disinfectant would avoid halogenation. There is scarcely any data published on the effects of ClO2 in drinking water on human or animal health. The kinetics of 36ClO2 was studied in rats. Radioactivity was rapidly absorbed from the gastrointestinal tract following the administration of (0.07 microCi) 36ClO2 orally. 36Cl in plasma reached at peak at 1 hr. The half life for the elimination of 36Cl from the rat was 44 hr, corresponding to a rate constant of 0.016 hr-1. After 72 hr radioactivity was highest in plasma, followed by kidney, lung, stomach, duodenum, ileum, liver, spleen, thymus, and bone marrow. 36Cl excretion was greatest at 24 and 48 hrs after the administration of 36 ClO2. Forty-three percent of the total initial dose was excreted at 72 hr in the urine and feces. No 36 Cl was detected in expired air throughout the 72 hr studied. ClO2, ClO2-, and ClO3- (1, 10, 100, 1000 ppm) given daily in drinking water decreased blood glutathione, decreased osmotic fragility, and changed the morphology of erythrocytes in both chicken and rat after two months. Methemoglobin was not detected throughout these studies.

PMID: 547024 [PubMed - indexed for MEDLINE]

Article
Heffernan WP, Guion C, Bull RJ.
Oxidative damage to the erythrocyte induced by sodium chlorite, in vivo.
Sodium chlorite in drinking water was found to produce a slight but compensated anemia in rats after exposure to up to 500 ppm for 90 days. Decreases in hemoglobin, red cell count, and packed cell volume seen after 30 days exposure had substantially recovered by 90 days of treatment. Signs of adaptation remained in that 2,3-diphosphoglyceric acid concentrations in the red cell remained elevated after 90 days exposure to 50 and 100 ppm CIO2-. However, dose-related decreases in erythrocyte glutathione levels, detected at chlorite levels as low as 50 ppm, remained decreased after 90 days exposure. While no other signs of overt toxicity were observed, the fact that hemolytic anemia was involved was confirmed by an increased turnover of red cells in cats exposed to CIO2-. Chlorite-induced decreases in glutathione in vivo were demonstrated to enhance formation of hydrogen peroxide when treated further with chlorite in vitro. Consequently, before a comprehensive determination of the hazards of chlorite in water can be made, particular attention must be paid to individuals sensitive to hemolytic anemia.

PMID: 528853 [PubMed - indexed for MEDLINE]

Article
Moore GS, Calabrese EJ.
The effects of chlorine dioxide and sodium chlorite on erythrocytes of A/J and C57L/J mice.
Because chlorinated surface drinking water supplies have been implicated in an increased risk of cancer, alternative methods of disinfection are being proposed; chlorine dioxide is the most seriously considered. This study reports that chlorine dioxide exposure of two strains of laboratory mice (A/J and C57L/J) to 100 ppm chlorine dioxide in their drinking water for 30 days produced no changes in 11 hematological parameters measured. Chlorite (a product formed from chlorine dioxide disinfection) produced increases in MCV (mean corpuscular volume); osmotic fragility; G6PD (glucose-6-phosphate dehydrogenase) activity; and the number of acanthocytes at exposure to 100 ppm, but not 1.0 or 10.0 ppm. These findings are consistent with membrane damage to the red cell and, in particular, the lipid fraction. Since chlorite is formed at a rate of 50 percent of the chlorine dioxide demand, serious consideration must be given to limiting chlorite formation before chlorine dioxide is adopted as a disinfectant to replace chlorine.

PMID: 7462915 [PubMed - indexed for MEDLINE]

Article
Moore GS, Calabrese EJ, Ho SC.
Groups at potentially high risk from chlorine dioxide treated water.
Chlorite, a by-product of chlorine dioxide disinfection of water, is a strong oxidant compound that produces markedly exaggerated effects in vitro on red cells of G6PD deficient humans when compared to normal human cells. Levels of methemoglobin are significantly greater and GSH levels significantly lower in the G6PD deficient cells than in normal cells after chlorite exposure. Persons with G6PD deficiency may be 3 to 4 times more likely to develop hemolytic anemia from chlorite exposure as persons with normal activity levels when GSH levels are used as a measure of susceptibility. The proposed use of chlorine dioxide as an alternate disinfectant for drinking water supplies should consider this potential high risk group.

PMID: 7462914 [PubMed - indexed for MEDLINE]

Article
Bercz JP, Jones L, Garner L, Murray D, Ludwig DA, Boston J.
Subchronic toxicity of chlorine dioxide and related compounds in drinking water in the nonhuman primate.
Subchronic toxicities of ClO2, NaClO2, NaClO3 and NH2Cl were studied in the African Green monkeys (Cercopithecus aethiops). The chemicals were administered in drinking water during 30-60 days subchronic rising dose protocols. The only unexpected and significant toxic effect was elicited by ClO2; this chemical inhibited thyroid metabolism in the animals at a dose of ca. 9.0 mg/kg/day. A statistically significant decrease of serum thyroxine occurred after the fourth week of exposure to 100 mg/l.concentration. The extent of thyroid suppression was dose dependent in each individual monkey, and was reversible after cessation of exposure. NaClO2 and NaClO3 failed to elicit similar effects in doses up to ca. 60 mg/kg/day. Also, NaClO4 or NH2Cl did not cause T-4 suppression in doses of 10 mg/kg/day. The selective thyroid effect of ClO2 was unexplained and it appeared to be paradoxical since ClO2 was rapidly reduced by the oral and gastric secretions to nonoxidizing species (presumably Cl-). No evidence of thyroid effects were detected in the serum of human volunteers who ingested approximately 1 mg/l. of ClO2 in drinking water as a result of routine use in the community water treatment process. Sodium chlorite induced dose-dependent oxidative stress on hematopoesis, causing decreased hemoglobin and red cell count and increased methemoglobin content. At the same time, serum transaminase (SGPT) levels showed significant subclinical elevation. The hematologic effects of NaClO2 rebounded during exposure indicating compensatory hemopoietic activity taking effect during oxidative stress. Sodium chlorate and chloramine did not induce detectable hematologic changes in the animals.

PMID: 7151767 [PubMed - indexed for MEDLINE]

Article
Couri D, Miller CH Jr., Bull RJ, Delphia JM, Ammar EM.
Assessment of maternal toxicity, embryotoxicity and teratogenic potential of sodium chlorite in Sprague-Dawley rats.
Groups of up to 13 pregnant rats were individually caged. bodyweight, food and water consumption were recorded at days 1, 8, 15 and 22 of gestation and the dams were treated on days 8-15 with sodium chlorite, 0.1%, 0.5% or 2% in drinking water or by injection of 10, 20, or 50 mg/kg IP or by gavaging with 200 mg/kg. To prevent ingestion of stillborn pups some dams were sacrificed at day 22. Other dams were allowed to deliver at term. Fetuses were weighed, measured and examined for soft tissue and skeletal malformations. Sodium chlorite, 20 or 50 mg/kg daily IP or gavaging with 200 mg/kg, caused vaginal and urethral bleeding. Doses of 10, 20 or 50 mg/kg daily IP caused 0, 50 and 100% mortality of dams, respectively. No deaths were caused by sodium chlorite in the drinking water, but the dams’ bodyweight, water and food consumption decreased during all treatments except 0.1% in the drinking water. Blood smears from the dams injected IP or drinking 2% sodium chlorite showed irregular, bizarre and ruptured erythrocytes. Injection of 10 or 20 mg/kg or drinking 2% resulted in decreased litter size and increased..